=====Palpitations=====
^'Traffic light system' for risk stratification of palpitations ||
^ • skipped beats\\ • thumping beats\\ • short fluttering\\ • slow pounding AND\\ • normal ECG AND\\ • No FHx AND\\ • no structural disease ^ • Hx suggests recurrent tachyarrhythmia\\ • palpitations with assoc symptoms AND/OR\\ • abnormal ECG AND/OR\\ • structural heart disease ^ • palpitations during exercise\\ • palpitations with syncope/near syncope\\ • high risk structural heart disease\\ • FHx of inheritable heart disease/SADS\\ • high degree AV block |
^ECG findings ^ Implication/consideration |
^Pre-excitation/delta wave |WPW – AVRT |
^Left atrial enlargement, frequent PACs, sinus bradycardia |[[wiki:cardiovascular:af|Atrial fibrillation]] |
^Left ventricular hypertrophy |[[wiki:cardiovascular:af|Atrial fibrillation]], ventricular tachycardia |
^Frequent PVCs |Ventricular tachycardia |
^Q waves |Ischaemic heart disease – [[wiki:cardiovascular:af|Atrial fibrillation]], ventricular tachycardia |
^Widespread T wave inversion across precordial leads,\\ LVH, Q waves and ST-segment changes |Hypertrophic cardiomyopathy – risk of atrial fibrillation, ventricular tachycardia |
^Long or short QT interval, Brugada pattern,\\ early repolarisation pattern |Genetic arrhythmia syndromes – risk of sudden cardiac death |
^Inverted T waves or Epsilon waves across right precordial leads (V1–V3)* |[[wiki:cardiovascular:arrhythmogenic_rv_dysplasia#arrhythmogenic_right_ventricular_cardiomyopathy|ARVC]] – risk of sudden cardiac death |
|//*in patients without RBBB//\\ ARVC - Arrhythmogenic Right Ventricular Cardiomyopathy ||
====Idiopathic Fascicular VT====
[[https://litfl.com/idiopathic-fascicular-left-ventricular-tachycardia/|LITFL - idiopathic fascic VT]]\\
[[https://www.escardio.org/Journals/E-Journal-of-Cardiology-Practice/Volume-9/Idiopathic-fascicular-left-ventricular-tachycardia|European Soc Cardiol idiopathic fascic VT 2010]]\\
*usually in young healthy adults 15-40yo, predominantly male resulting from ventricular ectopic focus
*a re-entrant tachy causing palpitations usually at rest but may be with exercise, stress, other catecholamine triggers etc
*often mis-diagnosed as SVT - look for dissociated P waves and narrow complex 'capture beat'
*good prognosis
***Monomorphic ventricular tachycardia** eg. fusion complexes, AV dissociation, capture beats
*90% of monomorphic VT = result of structural cardiac disease, usually IHD and others etc
*10% therefore NOT assoc with structural disease & therefore known as idiopathic VT with >75% of these arising in RV and the remainder in the L with most of these in L posterior fascicle
*QRS duration 100 – 140ms — ie. narrower than other forms of VT
*Short RS interval (onset of R to nadir of S wave) of 60-80 ms — the RS interval is usually > 100 ms in other types of VT
*RBBB Pattern
*Axis deviation depending on anatomical site of re-entry circuit (see classification)
*first line treatment = Verapamil
*Classification:
*Posterior fascicular VT (90-95%): RBBB morphology + left axis deviation; arises close to the left posterior fascicle
*Anterior fascicular VT (5-10%): RBBB morphology + right axis deviation; arises close to the left anterior fascicle
*Upper septal fascicular VT (rare): atypical morphology – usually RBBB but may resemble LBBB instead; cases with narrow QRS and normal axis have also been reported. Arises from the region of the upper septum
==Management==
*characteristically responds to verapamil
*does not respond to adenosine, vagal manoeuvres, lignocaine
==References include:==
https://bjcardio.co.uk/2009/07/10-steps-before-your-refer-for-palpitations/\\
https://www1.racgp.org.au/ajgp/2019/april/approach-to-palpitations\\