Palpitations

'Traffic light system' for risk stratification of palpitations

• skipped beats
• thumping beats
• short fluttering
• slow pounding AND
• normal ECG AND
• No FHx AND
• no structural disease

• Hx suggests recurrent tachyarrhythmia
• palpitations with assoc symptoms AND/OR
• abnormal ECG AND/OR
• structural heart disease

• palpitations during exercise
• palpitations with syncope/near syncope
• high risk structural heart disease
• FHx of inheritable heart disease/SADS
• high degree AV block

ECG findings

Implication/consideration

Pre-excitation/delta wave WPW – AVRT
Left atrial enlargement, frequent PACs, sinus bradycardia Atrial fibrillation
Left ventricular hypertrophy Atrial fibrillation, ventricular tachycardia
Frequent PVCs Ventricular tachycardia
Q waves Ischaemic heart disease – Atrial fibrillation, ventricular tachycardia
Widespread T wave inversion across precordial leads,
LVH, Q waves and ST-segment changes
Hypertrophic cardiomyopathy – risk of atrial fibrillation, ventricular tachycardia
Long or short QT interval, Brugada pattern,
early repolarisation pattern
Genetic arrhythmia syndromes – risk of sudden cardiac death
Inverted T waves or Epsilon waves across right precordial leads (V1–V3)* ARVCrisk of sudden cardiac death
*in patients without RBBB
ARVC - Arrhythmogenic Right Ventricular Cardiomyopathy

Idiopathic Fascicular VT

LITFL - idiopathic fascic VT
European Soc Cardiol idiopathic fascic VT 2010

  • usually in young healthy adults 15-40yo, predominantly male resulting from ventricular ectopic focus
  • a re-entrant tachy causing palpitations usually at rest but may be with exercise, stress, other catecholamine triggers etc
  • often mis-diagnosed as SVT - look for dissociated P waves and narrow complex 'capture beat'
  • good prognosis
  • Monomorphic ventricular tachycardia eg. fusion complexes, AV dissociation, capture beats
  • 90% of monomorphic VT = result of structural cardiac disease, usually IHD and others etc
  • 10% therefore NOT assoc with structural disease & therefore known as idiopathic VT with >75% of these arising in RV and the remainder in the L with most of these in L posterior fascicle
  • QRS duration 100 – 140ms — ie. narrower than other forms of VT
  • Short RS interval (onset of R to nadir of S wave) of 60-80 ms — the RS interval is usually > 100 ms in other types of VT
  • RBBB Pattern
  • Axis deviation depending on anatomical site of re-entry circuit (see classification)
  • first line treatment = Verapamil
  • Classification:
    • Posterior fascicular VT (90-95%): RBBB morphology + left axis deviation; arises close to the left posterior fascicle
    • Anterior fascicular VT (5-10%): RBBB morphology + right axis deviation; arises close to the left anterior fascicle
    • Upper septal fascicular VT (rare): atypical morphology – usually RBBB but may resemble LBBB instead; cases with narrow QRS and normal axis have also been reported. Arises from the region of the upper septum
Management
  • characteristically responds to verapamil
  • does not respond to adenosine, vagal manoeuvres, lignocaine
References include:
wiki/cardiovascular/palpitations.txt · Last modified: 2023/04/25 14:16 by 127.0.0.1
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